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In this study we examine whether and why daily work stressors are associated with sleep quality and sleep quantity. We draw on the effort-recovery model to test daily relationships between challenge and hindrance work stressors and sleep quality and quantity through physical and psychological fatigue, and vigor. We analyze daily diary data from 98 working sole mothers collected over seven days. The within-person daily hypotheses linking challenge and hindrance stressors to sleep via energetic mediators in our model were not supported. Exploratory analysis revealed several of our hypotheses were supported at the between-person level. Challenge and hindrance stressors were differentially related to psychological fatigue and physical fatigue such that hindrance stressors were positively associated, and challenge stressors were negatively associated. Challenge and hindrance stressors were differentially related to vigor such that hindrance stressors were negatively associated, and challenge stressors were positively associated. Across individuals, challenge and hindrance stressors were indirectly related to sleep quantity through these energetic mediators. This study answers calls for more investigations into mechanisms linking work stressors and sleep, and emphasizes the importance of examining phenomena at multiple levels of analysis. Theoretical and practical implications for the challenge-hindrance framework and effort-recovery model, including the appropriate timeframe for study, are discussed. 

Working sole mothers (i.e., non-partnered women who work) may experience elevated family demands that impose barriers to pursuing health behaviors during their daily leisure time. We aimed to map the process through which evening family demands influence leisure-time health behaviors in this priority population of employees, in an effort to identify targets for intervention development and health disparity reduction. Conducting a seven-day daily survey study in a sample of 102 working sole mothers, we supported perceptions of control over leisure time as a key mechanism linking evening family demands to leisure-time exercise. Furthermore, we identified the individual difference of present-focus (i.e., a tendency to focus on current experiences) as a key factor that alters how evening family demands affect control over leisure time, which ultimately mitigates the detrimental influence of these demands on evening exercise engagement. In contrast, we did not find evidence to support relationships of evening family demands with the health behaviors of leisure time consumption of alcohol or high sugar, high fat foods via control over leisure time. We discuss how our findings advance theory regarding how family demands influence health and inform practical efforts to reduce health disparities that working sole mothers face. 

Abstract Age-related reductions in neural selectivity have been linked to cognitive decline. We examined whether age differences in the strength of retrieval-related cortical reinstatement could be explained by analogous differences in neural selectivity at encoding, and whether reinstatement was associated with memory performance in an age-dependent or an age-independent manner. Young and older adults underwent fMRI as they encoded words paired with images of faces or scenes. During a subsequent scanned memory test participants judged whether test words were studied or unstudied and, for words judged studied, also made a source memory judgment about the associated image category. Using multi-voxel pattern similarity analyses, we identified robust evidence for reduced scene reinstatement in older relative to younger adults. This decline was however largely explained by age differences in neural differentiation at encoding; moreover, a similar relationship between neural selectivity at encoding and retrieval was evident in young participants. The results suggest that, regardless of age, the selectivity with which events are neurally processed at the time of encoding can determine the strength of retrieval-related cortical reinstatement.